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IMAGE: Increased levels of annexin A1, lactotransferrin, and aminopeptidase N. Increase the counts of anti-inflammatory macrophages with high motility and phagocytic ability, which increase repair of damaged tissue. Induces regulatory T. view more
Credit: Niigata University
Niigata, Japan - Researchers from Niigata University , the University of Tokyo, Osaka University and Tokyo Medical University, Japan have developed a new approach that could revolutionize the treatment, prevention, and possibly reversal of the damages caused by liver diseases. This novel strategy exploits small extracellular vesicles (sEVs) derived from interferon-γ (IFN-γ) pre-conditioned MSCs (γ-sEVs).
Cirrhosis and other chronic liver diseases remain a global health concern, with close to 2 million deaths reported annually, these account for approximately 3.5% of annual worldwide deaths. All these statistics are largely driven by the fact that 75 million of the 2 billion people who c
Study discovers mechanism involved in the development of metaplasia in the esophagus news-medical.net - get the latest breaking news, showbiz & celebrity photos, sport news & rumours, viral videos and top stories from news-medical.net Daily Mail and Mail on Sunday newspapers.
Study reveals the mechanisms involved in the transdifferentiation of esophageal keratinocytes into columnar cells
Metaplasia is defined as the replacement of a fully differentiated cell type by another. There are several classical examples of metaplasia, one of the most frequent is called Barrett s esophagus. Barrett s esophagus is characterized by the replacement of the keratinocytes by columnar cells in the lower esophagus upon chronic acid reflux.
This metaplasia is considered a precancerous lesion that increases by around 50 times the risk of this oesophageal adenocarcinoma. Nonetheless, the mechanisms involved in the development of metaplasia in the esophagus are still partially unknown.
In a new study published in
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Metaplasia is defined as the replacement of a fully differentiated cell type by another. There are several classical examples of metaplasia, one of the most frequent is called Barrett s oesophagus. Barrett s oesophagus is characterized by the replacement of the keratinocytes by columnar cells in the lower oesophagus upon chronic acid reflux. This metaplasia is considered a precancerous lesion that increases by around 50 times the risk of this oesophageal adenocarcinoma. Nonetheless, the mechanisms involved in the development of metaplasia in the oesophagus are still partially unknown.
In a new study published in
Cell Stem Cell, researchers led by Mr. Benjamin Beck, (FNRS research associate and WELBIO investigator at the IRIBHM, Université libre de Bruxelles, Belgium), report the mechanisms involved in the transdifferentiation of oesophageal keratinocytes into columnar cells.
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