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IMAGE: Ribosomes (large, dark circles) working their way down a strand of mRNA. Proteins are forming to the top and bottom. Amino acid scarcity results in ribosome logjams view more
Credit: the Weizmann Institute of Science
Cancers like melanoma are hard to treat, not least because they have a varied bag of tricks for defeating or evading treatments. A combined research effort by scientists at the Weizmann Institute of Science and researchers in the Netherlands Cancer Institute in Amsterdam and the University of Oslo, Norway, shows exactly how tumors, in their battles to survive, will go so far as to starve themselves in order to keep the immune cells that would eradicate them from functioning.
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BC Platforms delivers data platform for UK research consortium to improve detection of lung cancer
ZURICH and LONDON, Dec. 18, 2020 /PRNewswire/ BC Platforms, a global leader in clinical and genomic data management, analytics and access, today announced it is providing its discovery and research platform BC|INSIGHT to enable ground-breaking medical research into the early diagnosis of lung cancer in order to save lives. Lung cancer is the biggest cause of cancer death in the UK and worldwide.
The consortium is led by the Universities of Southampton and Leeds, together with healthcare, diagnostics and informatics companies, to test the best way of detecting cancers at a stage when they can still be cured, linking to the NHS England Lung Health Checks programme. In addition to BC Platforms, the research collaborators include Johnson & Johnson, Roche, Oncimmune, Inivata and others.
Study shows how melanomas assist T cells from identifying and killing tumor cells
Cancers like melanoma are hard to treat, not least because they have a varied bag of tricks for defeating or evading treatments.
Now, a combined research effort by the Weizmann Institute of Science, the Netherlands Cancer Institute in Amsterdam, and the University of Oslo shows exactly how tumors, in their fight to survive, will go so far as to starve themselves in order to keep the immune cells that would eradicate them from functioning. The work was published in
Nature.
The immunotherapies currently administered for melanomas work by removing obstacles that keep immune cells called T cells from identifying and killing tumor cells. Recent research suggested that, in melanoma, another blocker – one that stops IDO1, an enzyme that is overproduced by cancer cells – could also assist the T cells.
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