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Contraception blood-clot risk: ‘public need better access to advice’ Nicola Davis Science correspondent © Provided by The Guardian Photograph: Alamy Stock Photo
Women need more information about contraceptive options, experts said, after concerns over rare blood clots linked to the AstraZeneca Covid jab prompted a debate over side-effects caused by certain forms of the pill.
On Wednesday the UK’s Medicines and Healthcare products Regulatory Agency (MHRA) said that evidence that the jab could be causing a rare blood clotting syndrome was growing stronger. As a result the UK’s Joint Committee on Vaccination and Immunisation (JCVI) recommended that healthy people under the age of 30 who were at low risk of Covid should be offered a different vaccine if possible.
THE benefits of the Oxford/AstraZeneca vaccine continue to outweigh any risks, according to the UK medicines regulator, though under-30s will be offered an alternative jab. That vote of confidence has been backed up by the European medicines watchdog which ruled that unusual blood clots were “very rare side effects” of the jab. Some other European countries have restricted the vaccine’s use in younger people following reports of cerebral venous sinus thrombosis, a specific type of clot that prevents blood from draining from the brain, as well as low platelet counts – cells that help blood clot. However, Prime Minister Boris Johnson has said people should follow the regulator’s advice and keep getting their jabs.
Oxford-AstraZeneca COVID vaccine induces cell spikes similar to SARS-CoV-2 s
Scientists from the University of Oxford and the University of Southampton report in the journal
ACS Central Science that cells infected with the ChAdOx1 vaccine produce spike proteins on the cells similar to those produced by natural SARS-CoV-2 infection.
Artist imaging of protein spike on the surface of cells exposed to the vaccine. Image Credit: University of Southampton
The spike protein of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which protrudes from the virus envelope, is the key structure responsible for infecting host cells. The S1 subunit helps bind the virus to the angiotensin-converting enzyme 2 (ACE2), and the S2 subunit helps with membrane fusion with the host cell.
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