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Alzheimer s symptoms could be due to decline in brain protein not accumulation of amyloid plaques Experts estimate more than 6 million Americans are living with Alzheimer s dementia. But a recent study, led by the University of Cincinnati, sheds new light on the disease and a highly debated new drug therapy. The UC-led study, conducted in collaboration with the Karolinska Institute in Sweden, claims that the treatment of Alzheimer s disease might lie in normalizing the levels of a specific brain protein called amyloid-beta peptide. This protein is needed in its original, soluble form to keep the brain healthy, but sometimes it hardens into brain stones or clumps, called amyloid plaques.

United statesAlois alzheimerAlberto espayKariem ezzatEmily hendersonUniversity of cincinnatiKarolinska instituteAlzheimer disease neuroimaging initiativeUc gardner neuroscience instituteDisease neuroimaging initiativeஒன்றுபட்டது மாநிலங்களில்அலோயிஸ் முதுமறதிஎமிலி ஹென்டர்சன்பல்கலைக்கழகம் ஆஃப் ஸிந்ஸந்யாடீமுதுமறதி நோய் நியூரோஇமேஜிங் முயற்சிஅக் கார்ட்னர் நரம்பியல் நிறுவனம்