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Aging-US: Hyperbaric oxygen therapy impact on telomere length & immunosenescence

Thirty-five healthy independently living adults, aged 64 and older, were enrolled to receive 60 daily HBOT exposures. Whole blood samples were collected at baseline, at the 30th and 60th session, and 1-2 weeks following the last HBOT session. Telomeres length of T helper, T cytotoxic, natural killer and B cells increased significantly by over 20% following HBOT. In this Aging-US study, the most significant change was noticed in B cells which increased at the 30th session, 60th session and post HBOT by 25.68%±40.42 , 29.39%±23.39 and 37.63%±52.73, respectively. Dr. Amir Hadanny and Dr. Shai Efrati said, Aging can be characterized by the progressive loss of physiological integrity, resulting in impaired functions and susceptibility for diseases and death.

What is life? And will we find it on other planets? (video)

Aging-US: DNA- and telomere-damage does not limit lifespan: evidence from rapamycin

 E-Mail IMAGE: Rapamycin extends lifespan in natural but not progeroid mice. (A) Natural mice. Hyperfunctional aging (green/yellow/red arrow) progresses from development (green) to diseases (red), reaching death threshold and limiting lifespan. Accumulation. view more  Credit: Correspondence to: Mikhail V. Blagosklonny email: Blagosklonny@oncotarget.com Aging-US published DNA- and telomere-damage does not limit lifespan: evidence from rapamycin which reported that failure of rapamycin to extend lifespan in DNA repair mutant and telomerase-knockout mice, while extending lifespan in normal mice, indicates that neither DNA damage nor telomere shortening limits normal lifespan or causes normal aging. Dr. Mikhail V. Blagosklonny said, As a provocative title has recently announced, rapamycin fails to extend lifespan in DNA repair -deficient mice [1]. The word fails implies bad news. Rapamycin tried but failed. Yet, it is expected that the anti-aging drug rapamycin sho

Innovative flat optics will usher the next technological revolution

In a new paper published in Light: Science & Applications, the group led by Professor Andrea Fratalocchi from Primalight Laboratory of the Computer, Electrical and Mathematical Sciences and Engineering (CEMSE) Division, King Abdullah University of Science and Technology (KAUST), Saudi Arabia, introduced a new patented, scalable flat-optics technology manufactured with inexpensive semiconductors. The KAUST-designed technology leverages on a previously unrecognized aspect of optical nanoresonators, which are demonstrated to possess a physical layer that is completely equivalent to a feed-forward deep neural network. What we have achieved, explains Fratalocchi, is a technological process to cover flat surfaces, which in optical jargon are called flat optics, with physical neural units that are able to process light as a neural network does with an electrical signal.

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