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Study: Decreased activation of LEF1 gene disrupts ordinary neuronal function Lithium is considered the gold standard for treating bipolar disorder (BD), but nearly 70 percent of people with BD don t respond to it. This leaves them at risk for debilitating, potentially life-threatening mood swings. Researchers at the Salk Institute have found that the culprit may lie in gene activity or lack of it. A new study led by Salk Professor and President Rusty Gage, which published in the journal Molecular Psychiatry on January 4, 2021, shows that decreased activation of a gene called LEF1 disrupts ordinary neuronal function and promotes hyperexcitability in brain cells a hallmark of BD. The work could result in a new drug target for BD as well as a biomarker for lithium nonresponsiveness.

Renata santosCarol marchettoEmily hendersonSalk institute for biologicalSalk instituteStudy coResearch collaboratorSalk professorPresident rusty gageMolecular psychiatryStudy co first authorShani sternBipolar disorderValproic acidரெனாட்டா சாண்டோஸ்எமிலி ஹென்டர்சன்