Medical College of Wisconsin teams up with UW on $3 million study of state s health disparities biztimes.com - get the latest breaking news, showbiz & celebrity photos, sport news & rumours, viral videos and top stories from biztimes.com Daily Mail and Mail on Sunday newspapers.
In 2014, Dudley Lamming was reading a study out of Australia that had looked at how mice responded to dozens of controlled diets when one thing caught.
How low-protein diets might reprogram metabolism
In 2014, Dudley Lamming was reading a study out of Australia that looked at how mice responded to dozens of controlled diets when one thing caught his attention: The mice fed the least amount of protein were the healthiest.
“That was really interesting, because it goes against a lot of health information that people get,” says Lamming, a metabolism researcher in the University of Wisconsin School of Medicine and Public Health.
Since then, Lamming and graduate students in his laboratory have been trying to answer the question the Australian study raised: Why would low-protein diets make animals healthier?
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Can Restarting Aging Stem Cells Fight Memory Decline?
Researchers have discovered a mechanism linked to stem cell aging as well as how to reactivate the production of neurons.
By U. Zurich
As people get older, their neural stem cells lose the ability to proliferate and produce new neurons, leading to a decline in memory function.
The stem cells in our brain generate new neurons throughout life, for example in the hippocampus. This region of the brain plays a key role for a range of memory processes. With increasing age, and in patients suffering from Alzheimer’s disease, the hippocampus’ ability to create new neurons declines steadily and with it, its memory functions.
Metabolic switch may regenerate heart muscle following heart attack For news media
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Research from the University of Wisconsin–Madison finds that a new therapeutic approach for heart failure could help restore cardiac function by regenerating heart muscle.
In a study recently published in the journal Circulation, the UW team describes its success in improving, in a mouse model, the function of heart muscle by temporarily blocking a key metabolic enzyme after a heart attack. This simple intervention, the researchers say, could ultimately help people regain cardiac function.
“Our goal was to gain new understanding of how the heart can heal itself following injury at the molecular and cellular level and see if there was a way to restore cardiac function to an earlier state,” says UW–Madison’s Ahmed Mahmoud, professor of cell and regenerative biology in the School of Medicine and Public Health. “We know that a metabolic switch occurs in the heart f