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December 18, 2023
A new research paper was published on the cover of Aging (listed by MEDLINE/PubMed as "Aging (Albany NY)" and "Aging-US" by Web of Science) Volume 15, Issue 23, entitled, "Uncovering a unique pathogenic mechanism of SARS-CoV-2 omicron variant: selective induction of cellular senescence."
November 23, 2023
The cellular mechanisms of soy isoflavones, genistein, daidzein, and equol.
November 21, 2023
/PRNewswire/ -- The "Cyclin-Dependent Kinase Inhibitor - Pipeline Insight, 2023" drug pipelines has been added to ResearchAndMarkets.coms offering....
November 1, 2023
The study reveals that introducing progerin, a protein associated with accelerated aging, into cellular models of Alzheimer's Disease (AD) can hasten the development of AD-related traits. This innovative approach offers a more efficient method for studying AD mechanisms, potentially accelerating the discovery of new treatments.
September 24, 2023
Varanasi: A team of researchers from the School of Biotechnology, Institute of Science, Banaras Hindu University (BHU) has come up with a major findin.
September 23, 2023
Within the intricate molecular landscape inside of a cell, the orchestration of proteins demands precise control to avoid disease.
September 22, 2023
VARANASI : A team of researchers from the School of Biotechnology, Institute of Science, has come up with a major finding, which could be significantly important in exploring new therapy for cervical cancer. The team comprising Dr. Samarendra Kumar S
September 15, 2023
Shared genetic markers and immune system involvement in sJRA and T1D.
September 11, 2023
Researchers identify a super-enhancer-regulated RNA-binding protein cascade that drives the growth of pancreatic ductal adenocarcinoma, a deadly form of pancreatic cancer. The discovery offers a promising new avenue for targeted therapy, potentially sidestepping severe side effects associated with other treatments.
August 24, 2023
The current evidence on the role of the gut microbiome in metabolizing sweeteners and their metabolites.