Page 2 - Human Frontier Science Program News Today

<p>Professor Young Seok Ju's team unveiled a breakthrough study published in Cell Genomics that analyzed the whole-genome sequences of cells pre- and post-radiation exposure, pinpointing radiation-induced DNA mutations. Experiments involving cells from different organs of humans and mice exposed to varying radiation doses revealed mutation patterns correlating with exposure levels.</p>

Recent release of the waste water from Japan's Fukushima nuclear disaster stirred apprehension regarding the health implications of radiation exposure.

Cell death is fundamental to life and, thus, healthy aging. In the realm of cellular biology, ferroptosis (a form of programmed cell death) has emerged not only as a focal point of research for its potential in eliminating cancer cells, but also its role in a plethora of other diseases, including neurodegenerative diseases such as Alzheimer's disease, eye diseases such as Retinitis pigmentosa and age-related macular degeneration, as well as ischemia, cardiovascular disease, liver disease, ac...

Data gathered using software developed by Johns Hopkins University computer scientists will have 'huge implications' for understanding human health and evolution

A Ludwig Cancer Research study has generated a granular portrait of how the cellular and molecular components of the blood vessels that feed brain metastases of

A Ludwig Cancer Research study has generated a granular portrait of how the cellular and molecular components of the blood vessels that feed brain metastases of

A new study uncovers a molecular connection between vitamin B12 and multiple sclerosis (MS).

<p>Michigan State University researchers have shown that relationships and social interactions between hyenas influence when they “mob” lions.</p>

"If hyenas greet each other, they're more likely to mob. If they have strong social bonds, they're more likely to mob."

New MIT research shows that human microglia with the R47H/+ mutation in the TREM2 protein exhibit several deficits related to Alzheimer’s pathology. Mutant microglia are prone to inflammation, yet are worse at responding to neuron injury and less able to clear harmful debris, including amyloid beta.