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In an eight-year study of more than 600 community-dwelling older adults, Johns Hopkins Medicine researchers say they have further linked levels of cell-free DNA (DNA fragments resulting from cell death) circulating in the blood to chronic inflammation and frailty.

Scientists have discovered that a protein called neuritin may help some individuals resist Alzheimer’s disease and cognitive decline.

Neurodegenerative pathologies have shown few differences, but some cerebrovascular pathologies have decreased, while tau tangle levels have worsened over time.

Neurodegenerative pathologies have shown few differences, but some cerebrovascular pathologies have decreased, while tau tangle levels have worsened over time.

For adults over 65 years of age, higher systolic blood pressure is associated with an increased risk for dementia later in life.

Mount Sinai researchers have published a study in Alzheimer's & Dementia: The Journal of the Alzheimer's Association that sheds new light on the role of DNA methylation in Alzheimer's disease (AD).

For adults over 65 years of age, higher systolic blood pressure is associated with an increased risk for dementia later in life.

MIT research shows that in people carrying the APOE4 risk variant, a key brain cell type mismanages cholesterol needed to insulate neurons properly — another sign that APOE4 contributes to Alzheimer's disease by disrupting lipids in the brain.

When people carry the Alzheimer's associated APOE4 gene, oligodendrocytes fail to transport fatty molecules to wrap neurons that make brain circuitry connections. This deficit of myelin may contribute to the pathology and symptoms of Alzheimer's disease.

The overexpression of a gene tied to cell division and the structure and function of neurons may prevent and protect against cognitive decline in both mice and humans with Alzheimer’s disease (AD), according to a new study by scientists at the University of Colorado Anschutz Medical Campus.