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The Identity Crisis of Alzheimer s Brain Cells
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Salk scientists reveal how brain cells in Alzheimer s go awry, lose their identity
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The walking tours begin at the Athenaeum and weave through The Village, covering roughly half the murals currently on view, including the five installed in 2020 and 2021. “Each mural has its own story,” Forsha said. “We talk about the artists’ concepts, what the inspiration might have been.”
The tours are free and will be limited at first to 20 of the usual 40-person capacity, with masks required.
“I’ve really missed giving the tours,” Forsha said. “I love spending time with people who are just curious. I find that the people who sign up for the tours are always interested in knowing more about the project.”
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IMAGE: In this image of a neuron nucleus, bright spots show areas of focused genetic repair. view more
Credit: Salk Institute/Waitt Advanced Biophotonics Center
LA JOLLA (April 1, 2021) Neurons lack the ability to replicate their DNA, so they re constantly working to repair damage to their genome. Now, a new study by Salk scientists finds that these repairs are not random, but instead focus on protecting certain genetic hot spots that appear to play a critical role in neural identity and function.
The findings, published in the April 2, 2021, issue of
Science, give novel insights into the genetic structures involved in aging and neurodegeneration, and could point to the development of potential new therapies for diseases such Alzheimer s, Parkinson s and other age-related dementia disorders.
Salk scientist Ronald Evans receives 2021 Asan Award in Basic Medicine
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