Kinesin-5 Gene May Protect Against Alzheimer's Disease
The gene Kinesin-5 or KIF11 accomplishes this despite the fact that amyloid beta, the main component of plaques in the brains of those with AD, is present. In
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The gene Kinesin-5 or KIF11 accomplishes this despite the fact that amyloid beta, the main component of plaques in the brains of those with AD, is present. In
Risk models based on noncognitive measures may predict presymptomatic Alzheimer's disease.
A new research paper was published in Aging (abbreviated as "Aging (Albany NY)" by Medline/PubMed and as "Aging-US" by Web of Science) on the cover of Volume 14, Issue 14, entitled, "Aging the brain: multi-region methylation principal component based clock in the context of Alzheimer's disease."
Traumatic brain injury even without loss of consciousness may be an important risk factor for late-life cognitive impairment, new research suggests.
Fifty-two cognitive resilience proteins were specific for cognitive decline and an estimated 30% may offer resilience for both cognitive and noncognitive AD phenotypes.