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Protein may clarify Alzheimer's and body clock link


A brain protein may help explain the link between circadian rhythm and Alzheimer’s disease, researchers report.
Fractured sleep, daytime sleepiness, and other signs of disturbance in one’s circadian rhythm are common complaints of people with Alzheimer’s disease, and the problems only get worse as the disease progresses.
Researchers say that a clue may lie in the brain protein YKL-40.
The researchers report that clock genes regulate YKL-40 and it’s also involved in clearing away potentially toxic buildup of Alzheimer’s proteins in the brain. Moreover, Alzheimer’s patients who carry a genetic variant that reduces YKL-40 levels maintain their cognitive faculties longer than people without the variant, the scientists found. ....

Brianv Lananna , Erik Musiek , David Holtzman , Carlos Cruchaga , Anne Fagan , Joanne Knight Alzheimer Disease Research Center , Joanne Knight Alzheimer , Disease Research , டேவிட் ஹோல்ட்ஸ்மேன் , அன்னே ஃபாகன் , ஜொவந் நைட் முதுமறதி நோய் ஆராய்ச்சி மையம் , ஜொவந் நைட் முதுமறதி , நோய் ஆராய்ச்சி ,

Alzheimer's Disease Linked to Dysfunction Circadian Clock


Studies by a research team at Washington University School of Medicine in St. Louis indicate that a brain protein known as YKL-40 may link Alzheimer’s disease with dysfunction in circadian rhythms, suggesting that treatments that target the protein could slow the course of the disease. Their work, reported in
Science Translational Medicine, found that YKL-40 is both regulated by clock genes and involved in clearing away the potentially toxic build-up of Alzheimer’s proteins in the brain. The team’s studies indicated that Alzheimer’s patients who carry a genetic variant that reduces YKL-40 levels maintain their cognitive faculties longer than those individuals without the variant. ....

Brianv Lananna , Erik Musiek , David Holtzman , Carlos Cruchaga , Gretchenp Jones , Anne Fagan , Joanne Knight Alzheimer Disease Research Center , Washington University School Of Medicine , Washington University School , Science Translational Medicine , Joanne Knight Alzheimer , Disease Research , டேவிட் ஹோல்ட்ஸ்மேன் , அன்னே ஃபாகன் , ஜொவந் நைட் முதுமறதி நோய் ஆராய்ச்சி மையம் , வாஷிங்டன் பல்கலைக்கழகம் பள்ளி ஆஃப் மருந்து , வாஷிங்டன் பல்கலைக்கழகம் பள்ளி , அறிவியல் மொழிபெயர்ப்பு மருந்து , ஜொவந் நைட் முதுமறதி , நோய் ஆராய்ச்சி ,

Brain protein indicates a link between circadian rhythm dysfunction and Alzheimer's Disease


Brain protein indicates a link between circadian rhythm dysfunction and Alzheimer s Disease
Fractured sleep, daytime sleepiness and other signs of disturbance in one s circadian rhythm are common complaints of people with Alzheimer s disease, and the problems only get worse as the disease progresses. But the reason for the link between Alzheimer s and circadian dysfunction is not well understood.
Researchers at Washington University School of Medicine in St. Louis say that a clue may lie in the brain protein YKL-40.
In a study published Dec. 16 in Science Translational Medicine, the researchers report that YKL-40 is both regulated by clock genes and involved in clearing away potentially toxic buildup of Alzheimer s proteins in the brain. Moreover, Alzheimer s patients who carry a genetic variant that reduces YKL-40 levels maintain their cognitive faculties longer than people without the variant, the scientists found. ....

Brianv Lananna , Erik Musiek , David Holtzman , Carlos Cruchaga , Gretchenp Jones , Anne Fagan , Emily Henderson , Researchers At Washington University School Of Medicine , Department Of Neurology , Joanne Knight Alzheimer Disease Research Center , Washington University In St , Washington University School , Science Translational Medicine , Study Senior Author , Associate Professor , Washington University , Joanne Knight Alzheimer , Disease Research , டேவிட் ஹோல்ட்ஸ்மேன் , அன்னே ஃபாகன் , எமிலி ஹென்டர்சன் , ஆராய்ச்சியாளர்கள் இல் வாஷிங்டன் பல்கலைக்கழகம் பள்ளி ஆஃப் மருந்து , துறை ஆஃப் நரம்பியல் , ஜொவந் நைட் முதுமறதி நோய் ஆராய்ச்சி மையம் , வாஷிங்டன் பல்கலைக்கழகம் இல் ஸ்டம்ப் , வாஷிங்டன் பல்கலைக்கழகம் பள்ளி ,

Protein involved in removing Alzheimer's buildup linked to circadian rhythm | The Source


Brain protein helps explain link between circadian rhythm, Alzheimer’s disease
December 16, 2020
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Immune cells known as microglia (turquoise with red dots) surround a plaque of the Alzheimer’s protein amyloid (blue). The red dots indicate that the microglia are prepared to remove the potentially damaging plaque. Researchers at Washington University School of Medicine in St. Louis have discovered a protein that links the amyloid-removal process to the circadian clock. The protein, YKL-40, could help explain why people with Alzheimer’s frequently suffer from sleep disturbances, and provide a new target for Alzheimer’s therapies. (Image: Brian Lananna/School of Medicine) ....

Brianv Lananna , Erik Musiek , David Holtzman , Gretchenp Jones , Carlos Cruchaga , Anne Fagan , Brian Lananna School Of Medicine , Researchers At Washington University School Of Medicine , Department Of Neurology , Joanne Knight Alzheimer Disease Research Center , Washington University School , Science Translational Medicine , Joanne Knight Alzheimer , Disease Research , டேவிட் ஹோல்ட்ஸ்மேன் , அன்னே ஃபாகன் , ஆராய்ச்சியாளர்கள் இல் வாஷிங்டன் பல்கலைக்கழகம் பள்ளி ஆஃப் மருந்து , துறை ஆஃப் நரம்பியல் , ஜொவந் நைட் முதுமறதி நோய் ஆராய்ச்சி மையம் , வாஷிங்டன் பல்கலைக்கழகம் பள்ளி , அறிவியல் மொழிபெயர்ப்பு மருந்து , ஜொவந் நைட் முதுமறதி , நோய் ஆராய்ச்சி ,

Protein involved in removing Alzheimer's buildup linked to circadian rhythm