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Scientists uncover evolutionary forces at play in aging of blood system and identify people
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Differential regulation of G protein signaling in Arabidopsis through two distinct pathways that internalize AtRGS1
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SARS-CoV-2 virus uses a multipronged strategy to avoid detection by the immune system
Severe symptoms of COVID-19, leading often to death, are thought to result from the patient s own acute immune response rather than from damage inflicted directly by the virus.
Intensive research efforts are therefore seeking to determine how the SARS-CoV-2 virus manages to mount an effective invasion while throwing the immune system off course. A new study, published in
Nature, reveals a multipronged strategy that the virus employs to ensure its quick and efficient replication, while avoiding detection by the immune system.
The study, conducted jointly by the research groups of Dr. Noam Stern-Ginossar at the Weizmann Institute of Science and Dr. Nir Paran and Dr. Tomer Israely of the Israel Institute for Biological, Chemical and Environmental Sciences, focused on understanding the molecular mechanisms at work during infection by SARS-CoV-2 at the cellular level.
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