Blocking Notch signaling pathway could reverse age-related b

A cellular signal essential to the development of the skeleton increases during aging to weaken bones, finds a new study in mice.

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Germany , New York , United States , Christopher Park , Aristotelis Tsirigos , Ralf Adams , Philipp Leucht , Lukasz Witek , Mohamed Ali , Danielle Ellis , Backialakshmi Dharmalingam , Sophie Morgani , Paulo El Parente , Vasudev Nayak , Malissa Ramsukh , Ziyan Lin , Lauren Gillinov , Kevin Leclerc , Alesha Castillo , Sooyeon Lee , National Institutes Of Health , Langone Health , York Stem Cell Foundation , York University College Of Dentistry , Max Planck Institute For Molecular Biomedicine , Bone Research , Department Of Molecular Pathobiology , Department Of Pathology , Department Of Tissue Morphogenesis , Department Of Orthopedic Surgery , Grossman School Of Medicine , Isaac Perlmutter Cancer Center , Raj Sobti Menon Associate Professor , Orthopedic Surgery , Cell Biology , Lindsey Remark , Margaux Sambon , Pablo Atria , Applied Bioinformatics Laboratories , Molecular Pathobiology , New York University College , Max Planck Institute , Molecular Biomedicine , Tissue Morphogenesis , National Institutes , Frank Zarb Family , New York Stem Cell , Aging , One , Fell , Adipocytes , Bone Marrow , Dancer , Fracture , Genes , Enetic , Medicine , Notch Signaling , Rthopedic , Research , Na , Ignaling Pathway , Stem Cells , Surgery ,