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Modulating BST2 expression on blood stem cells may help regulate cell activation during chronic infections

Modulating BST2 expression on blood stem cells may help regulate cell activation during chronic infections
When a person has an infection, the body activates immune responses to fight it. IFNγ is an inflammatory molecule produced by the immune system that helps fight infections.
However, long-term exposure to IFNγ has undesirable consequences - it irreversibly exhausts blood stem cells, the progenitors of all blood cells, including immune cells, by triggering their proliferation and excessive terminal differentiation. Lacking immune cells, patients eventually are unable to fight infections.
Baylor College of Medicine researchers investigated the mechanism by which IFNγ mediates depletion of blood stem cells, which are normally quiescent, meaning they are in a dormant state until activated when stimulated. ....

Katherine King , Emily Henderson , Marcusa Florez , Study Co , Baylor College Of Medicine , Cell Reports , Danl Duncan Comprehensive Cancer Center , Dongsu Park , Study Co Corresponding Author , Associate Professor , Pediatrics Infectious Diseases , Baylor College , Medical Scientist Training Program , க்யாதரிந் கிங் , எமிலி ஹென்டர்சன் , படிப்பு இணை , பேலர் கல்லூரி ஆஃப் மருந்து , செல் அறிக்கைகள் , இணை ப்ரொஃபெஸர் , குழந்தை மருத்துவம் தொற்று நோய்கள் , பேலர் கல்லூரி , மருத்துவ விஞ்ஞானி பயிற்சி ப்ரோக்ர்யாம் ,

Too much of a good thing - persistent IFNγ depletes progenitor blood cells via BST2

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When a person has an infection, the body activates immune responses to fight it. IFNγ is an inflammatory molecule produced by the immune system that helps fight infections. However, long-term exposure to IFNγ has undesirable consequences - it irreversibly exhausts blood stem cells, the progenitors of all blood cells, including immune cells, by triggering their proliferation and excessive terminal differentiation. Lacking immune cells, patients eventually are unable to fight infections.
Baylor College of Medicine researchers investigated the mechanism by which IFNγ mediates depletion of blood stem cells, which are normally quiescent, meaning they are in a dormant state until activated when stimulated. They showed in the journal ....

Youngjae Jeong , Roman Jaksik , Laura Ortinau , Katie Matatall , Paul Shafer , Katherine King , Marcusa Florez , Marek Kimmel At Silesian University Of Technology , Rice University , Baylor College Of Medicine , Cell Reports , Danl Duncan Comprehensive Cancer Center , Dongsu Park , Medical Scientist Training Program , Anne Lynch , Marek Kimmel , Silesian University , பால் ஷாபெர் , க்யாதரிந் கிங் , அரிசி பல்கலைக்கழகம் , பேலர் கல்லூரி ஆஃப் மருந்து , செல் அறிக்கைகள் , மருத்துவ விஞ்ஞானி பயிற்சி ப்ரோக்ர்யாம் , அன்னே லிஞ்ச் , மரேக் கீம்மேல் ,

Too much of a good thing—persistent IFNγ depletes progenitor blood cells via BST2

Too much of a good thing—persistent IFNγ depletes progenitor blood cells via BST2
medicalxpress.com - get the latest breaking news, showbiz & celebrity photos, sport news & rumours, viral videos and top stories from medicalxpress.com Daily Mail and Mail on Sunday newspapers.

Katherine King , Marcusa Florez , Baylor College Of Medicine , Cell Reports , Danl Duncan Comprehensive Cancer Center , Dongsu Park , Medical Scientist Training Program , க்யாதரிந் கிங் , பேலர் கல்லூரி ஆஃப் மருந்து , செல் அறிக்கைகள் , மருத்துவ விஞ்ஞானி பயிற்சி ப்ரோக்ர்யாம் ,