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NU faculty research, educate for Autism Acceptance Month - Vimarsana News

NU faculty research, educate for Autism Acceptance Month

April marks the start of Autism Awareness and Acceptance Month, which is dedicated to promoting inclusion and understanding of autistic people. At Northwestern, faculty are continuing to make strides in both research and education to further understand ASD.  Communication Prof. Molly Losh, who teaches a course on autism and leads the Neurodevelopmental Disability Lab within...

Brain over-activation could be causing seizures and social deficits in genetic subtype of autism - Vimarsana News

Brain over-activation could be causing seizures and social deficits in genetic subtype of autism

Northwestern Medicine scientists have identified the cause of a genetic subtype of autism and schizophrenia that results in social deficits and seizures in mice and humans.

Social deficits, seizures in autism type linked to overactive brain circuits: Study - Vimarsana News

Social deficits, seizures in autism type linked to overactive brain circuits: Study

A genetic variant of autism and schizophrenia that causes social deficits and seizures in mice and people has been linked to a cause, according to researchers from Northwestern Medicine.

Science News | Social Deficits, Seizures in Autism Type Linked to Overactive Brain Circuits: Study - Vimarsana News

Science News | Social Deficits, Seizures in Autism Type Linked to Overactive Brain Circuits: Study

Get latest articles and stories on Science at LatestLY. A genetic variant of autism and schizophrenia that causes social deficits and seizures in mice and people has been linked to a cause, according to researchers from Northwestern Medicine. Science News | Social Deficits, Seizures in Autism Type Linked to Overactive Brain Circuits: Study.

Social Deficits and Seizures in Autism Type Tied to Overexcited Brain Circuits - Vimarsana News

Social Deficits and Seizures in Autism Type Tied to Overexcited Brain Circuits

A genetic subtype of ASD and schizophrenia have a duplicate gene that triggers overactivity in neural circuits, leading to social deficits and seizures. When researchers reduced the level of the PRRT2 gene in mouse models of 16p11.2 duplication syndrome, social behaviors were restored and seizure activity decreased.