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A team of Mount Sinai researchers has produced a high-resolution crystal structure of an enzyme essential to the survival of SARS-CoV-2, the virus that causes COVID-19.

Mount Sinai researchers have produced a high-resolution crystal structure of an enzyme essential to the survival of SARS-CoV-2, the virus that causes COVID-19.

Researchers have been studying zombie cells, aka senescent cells, and their link to aging. Could zombie cell research slow down aging in the future?

The protective caps at the ends of chromosomes naturally shorten over time and damage can trigger senescence and contribute to age-related diseases.

Senescent cells — those that have lost the ability to divide — accumulate with age and are key drivers of age-related diseases, such as cancer, dementia and cardiovascular disease. In a new study, a team led by University of Pittsburgh and UPMC Hillm

Regeneron Pharmaceuticals, Inc. (NASDAQ: REGN) today announced the winners of the 10th annual Regeneron Prize for Creative Innovation, a competition designed to recognize excellence and creativity in biomedical research conducted by postdoctoral fellows and graduate students. Each year, Regeneron invites the ...

Damage to the ends of your chromosomes can create “zombie cells” that are still alive but can’t function, according to our recently published study in Nature Structural and Molecular Biology. When cells prepare to divide, their DNA is tightly wound around proteins to&#...

Damage to the ends of your chromosomes can create "zombie cells" that are still alive but cant function, according to our recently published study in Nature Structural and Molecular Biology.

Zombie cells can be good for health by inviting immune cells to fight any cancer cells but can also trigger tumour growth

The protective caps at the ends of chromosomes naturally shorten over time. Researchers found that direct damage can prematurely trigger senescence and contribute to age-related diseases like cancer.