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Insilico Medicine collaborates with the Indiana Biosciences Research Institute to apply novel artificial intelligence-powered approaches to multi-omics data analysis and discovery of potential therapeutic targets. ....
New Curtin research has shown how a readily available, cheap and safe-to-use product found in the medicine cabinet of most homes could be the key to better ecological restoration practices with major benefits for the environment and agriculture.
The study revealed that aspirin, which naturally occurs in the bark of the willow tree and other plants, can improve the survival of grass species important for ecological restoration and sustainable pasture when applied in a seed coating. ....
Credit: Hanna Salmonowicz, Monney Medical Media, 2021 Our genetic material is stored in our cells in a specific way to make the meter-long DNA molecule fit into the tiny cell nucleus of each body cell. An international team of researchers at the Max Planck Institute for Biology of Ageing, the CECAD Cluster of Excellence in Ageing research at the University of Cologne, the University College London and the University of Michigan have now been able to show that rapamycin, a well-known anti-ageing candidate, targets gut cells specifically to alter the way of DNA storage inside these cells, and thereby promotes gut health and longevity. This effect has been observed in flies and mice. The researchers believe this finding will open up new possibilities for targeted therapeutic interventions against ageing. ....
A new study conducted by the researchers at the University of Liverpool reveals how the ancient photosynthetic organisms - cyanobacteria - evolve their photosynthetic machinery and organise their photosynthetic membrane architecture for the efficient capture of solar light and energy transduction. ....
E-Mail IMAGE: In Alg mutant embryos, rod cells are initially born but not maintained and undergo programmed cell death indicated in magenta (TUNEL staining). view more Credit: Clara Becker. Human cells are kept healthy by the activity of millions of proteins. These proteins are modified in different ways, such as by adding sugar molecules to them, which can be crucial for them to function properly. Given this importance, defects in the sugar-adding process are often lethal at the very early stages of development. In rare cases, however, patients can develop sugar-adding deficiencies that result in a range of metabolic diseases, known collectively as congenital disorders of glycosylation (CDG). These disorders are caused by defects in the enzymes involved in the sugar-adding process. For example, ALG2-CDG (or CDG-Ii) is a disorder caused by mutations in the ALG2 enzyme, which combines sugar molecules together. ALG2-CDG patients appear unaffected at bir ....