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Change in diet and circadian clock can impact healthy fat tissue, finds study


Change in diet and circadian clock can impact healthy fat tissue, finds study
Changing your eating habits or altering your circadian clock can impact healthy fat tissue throughout your lifespan, according to a preclinical study published today in
Nature by researchers with The University of Texas Health Science Center at Houston (UTHealth).
Healthy fat tissue helps provide energy, supports cell growth, protects organs, and keeps the body warm. A good quality diet and one that is consumed in a rhythmic manner (i.e., during our active cycle) is important in maintaining healthy fat, the researchers found.
Adipocyte progenitor cells mature into adipocytes - the healthy fat cells that make up our adipose tissue, which stores energy as fat. Researchers discovered that adipocyte progenitors undergo rhythmic daily proliferation throughout the 24-hour cycle under normal patterns of energy intake. ....

Kristin Eckel Mahan , Aleix Ribas Latre , Emily Henderson , Helmholtz Institute For Metabolic , University Hospital Leipzig , Vascular Research , University Of Leipzig , Texas Health Science Center , Degenerative Diseases , Brown Foundation Institute , Molecular Medicine , Human Diseases , Helmholtz Institute , கிறிஸ்டின் எக்கேள் மகான் , எமிலி ஹென்டர்சன் , பல்கலைக்கழகம் மருத்துவமனை லெயிஸீக் , வாஸ்குலர் ஆராய்ச்சி , பல்கலைக்கழகம் ஆஃப் லெயிஸீக் , டெக்சாஸ் ஆரோக்கியம் அறிவியல் மையம் , சீரழிவு நோய்கள் , பழுப்பு அடித்தளம் நிறுவனம் , மூலக்கூறு மருந்து , மனிதன் நோய்கள் ,

Aging, diet-induced obesity, and metabolic disease link explored in new research


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Unraveling the links among obesity, aging, telomere lengths and metabolic diseases is the subject of the study published today in
Nature Metabolism by a collaborative research team at The University of Texas Health Science Center at Houston (UTHealth).
Telomeres act as protective caps at the end of chromosomes to prevent them from replication errors during cell divisions. Every time a chromosome replicates itself, telomeres shorten. When the telomeres become too short, the cell can no longer replicate its chromosomes safely and becomes arrested, or senescent. That shortening has been linked to the aging process and development of degenerative diseases. ....

Yulin Dai , Brade Snyder , Mikhail Kolonin , Cale Fussell , Harrye Bovay Jr , Kristinl Eckel Mahan , Alexesc Daquinag , Zhongming Zhao , Zhanguo Gao , Angielyn Rivera , Metabolic Disease Research , Uthealth School Of Biomedical Informatics , Mcgovern Medical School , Nature Metabolism , Texas Health Science Center , Distinguished University Chair , Degenerative Diseases , Brown Foundation Institute , Molecular Medicine , Human Diseases , Mcgovern Medical , Biomedical Informatics , Graduate School , யூலின் தாய் , பிரேட் ஸ்னைடர் , காலே ஃபஸல் ,