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A new study from MIT shows how a type of cells called microglia contribute to the slowdown of neuron activity seen in Alzheimer’s disease. The study found that microglia that express the APOE4 gene, one of the strongest genetic risk factors for Alzheimer’s disease, cannot metabolize lipids normally, leading to a buildup of excess lipids that interferes with nearby neurons’ ability to communicate with each other.

New findings show how stem cells coordinate to repair and replace the intestine's lining. The work could clarify disorders like inflammatory bowel disease.

To act as a robust barrier against pathogens while also absorbing needed nutrients, the lining of the intestines must regenerate on a daily basis to remain equal to the task.

Press release content from Business Wire. The AP news staff was not involved in its creation.

-- Proceeds will enable Xilis to build its capabilities in supporting biopharma collaborations for drug discovery -- -- Extension led by new investor FPV Ventures -- Xilis, Inc., a pioneering biotechnology