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When people carry the Alzheimer's associated APOE4 gene, oligodendrocytes fail to transport fatty molecules to wrap neurons that make brain circuitry connections. This deficit of myelin may contribute to the pathology and symptoms of Alzheimer's disease.
“People should not say, ‘Oh, great, they’re making a pill; I can stop exercising now.’” Read more on Boston.com.
Researchers have mapped cells, genes, and cellular pathways that are modified by exercise or high-fat diet.
Researchers with MIT and Harvard Medical School conducted a study to see how exercise and high-fat diets impacted the cells of mice.
MIT and Harvard Medical School researchers mapped out many of the cells, genes, and cellular pathways that are modified by exercise or a high-fat diet. They hope their findings will help guide the design of drugs that might mimic some beneficial effects of exercise.