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Tanja Gruber wechselt von der AUA zu den Wiener Linien

Tanja Gruber wechselt von der AUA zu den Wiener Linien
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Risk-directed childhood leukemia treatment takes a step forward


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Risk-directed childhood leukemia treatment takes a step forward
Corresponding author Ching-Hon Pui, M.D., St. Jude Oncology Department chair, helped discover how genomic analysis can improve outcomes for children with acute lymphoblastic leukemia.
Comprehensive genomic analyses have helped researchers identify more than 20 subtypes of acute lymphoblastic leukemia (ALL) based on the genetic mutations that drive the disease. Research led by St. Jude Children’s Research Hospital scientists showed that combining these data with leukemia response measurements improves prediction of relapse risk.
Currently, patients’ response to treatment influences the type of therapy administered for ALL. This risk-directed approach involves assessing minimal residual disease, the level of leukemic cells in the blood or bone marrow of patients at defined points during treatment ....

Deqing Pei , Elaine Coustan Smith , Zhaohui Gu , Dario Campana , William Evans , Mary Relling , John Choi , Jeffrey Rubnitz , Sima Jeha , Tanja Gruber , Susana Raimondi , Charles Mullighan , Junj Yang , Kathryn Roberts , Seth Karol , Samuel Brady , Jamesr Downing , Raul Ribeiro , Hiroto Inaba , Jude Oncology Department , Judeoncology Department , Jude Children Research Hospital , National University Of Singapore , National Institutes Of Health , Jude Department Of Pathology , Ching Hon Pui ,

Study reveals mutations that drive therapy-related myeloid neoplasms in children


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IMAGE: Xiaotu Ma, Ph.D., St. Jude Computational Biology, and Jeffery Klco, M.D., Ph.D., St. Jude Pathology
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Credit: St. Jude Children s Research Hospital
Children treated for cancer with approaches such as chemotherapy can develop therapy-related myeloid neoplasms (a second type of cancer) with a dismal prognosis. Scientists at St. Jude Children s Research Hospital have characterized the genomic abnormalities of 84 such myeloid neoplasms, with potential implications for early interventions to stop the disease. A paper detailing the work was published today in
Nature Communications.
The somatic (cancer) and germline (inherited) genomic alterations that drive therapy-related myeloid neoplasms in children have not been comprehensively described, until now. The researchers used a variety of sequencing techniques (whole exome, whole genome and RNA) to characterize the genomic profile of 84 pediatric therapy-related myeloid neoplasm ....

United States , Jason Schwartz , Cristyn Branstetter , Scott Newman , Kim Nichols , Jing Ma , Ryan Hiltenbrand , Tamara Westover , Priyadarshini Kumar , Xiaolong Chen , Jennifer Kamens , John Easton , Jeffrey Rubnitz , Jeffery Klco , Xiaotu Ma , Tanja Gruber , Michael Walsh , Charles Mullighan , Jinghui Zhang , Samuel Brady , Lindsey Montefiori , J Robert Michael , Yanling Liu , Jamie Maciaszek , Huiyn Wu , Nature Communications ,